Important Principles of Diagnosis in Diabetic Nephropathy
Keywords:
gout, hyperuricemia, uric acid, arterial hypertension, microalbuminuria, diabetes, metabolic syndromeAbstract
Hyperuricemia is the result of metabolic diseases and is one of the most common problematic diseases in recent years. The most common manifestations of this phenomenon are gouty arthritis, the appearance and formation of urate kidney stones. The problem of hyperuricemia and gout is a classic model of this trend, only going beyond articular syndrome. The current concept of gout nephropathy combines different clinical manifestations of uric acid metabolism: from direct damaging effects on joints and kidney tissue to indirect lipid and carbohydrate metabolism defects (by including them in clinical syndromes).
References
1. Bergar L., Yu T. Renal changes in gout nephropathy // Am. J. Med. 1975. V. 59. P. 605-608
2. Vazguez-Mellado J., Garcia S.G., Vanguez S.G. et al. Metabolic syndrome and ischemic heart disease in gout nephropathy // J. Clinic. Revmatol. 2004. Volume 10 (3). P. 105-109.
3. Gersh M.S., Renal function in gout nephropathy // Am. J. Med. 2007. V.59. P. 605-608
4. Kanbay M., Metabolic pathways of high-density lipoprotein, low-density lipoprotein, and triglycerides: flow rewire. // Am. J. Cardiol. 2000. V. 86. (12 appendix L). P. 5-10
5. Kudasheva F.M., Eliseev M.S., Barskova V.G., Nasanova V.A. Comparison of the onset rate of analgesic and anti-inflammatory effects of different nimesulide and diclofenac sodium in gouty arthritis // Ter.archiv. 2007. № 5. S. 35-40
6. Lawrence RC, Helmisk CG, Arnett F.C. et al. Estimates of the prevalence of arthritis and selected musculoskeletal system in the United States.// Arthr. Revm. 2008. № 41. S. 778-99.
7. Muxin N.A., Balkarov N.M., Maksimov N.A. Clinical manifestations of disorders of purine metabolism in internship practice // Ter.archiv.- 1994.- №1. -FROM. 35-39.
8. Muxin N.A., Serov V.V., Maksimov NA. Some features of the hyperuricemic variant of occult glomerulonephritis. // Ter. Archive. 2000. № 6. P.43-46.d complications of the disease is a topical issue of therapy.
9. Nakagawa T., Mazalli M., Kang D-H. Et al. Hyperuricemia causes glomerular hypertrophy in rats. // Am. J. Nefrol. 2003. Volume 23. R. 2-7.
10. Hozla U.M., Jarikov S., Finchi J.L., Nokagawa T., Ronkal C., Mu V., Krotova K., Blosk E.R., Prabhakar S., Johnson R.J. Hyperuricemia causes endothelial dysfunction // Renal internation. 2005. Volume 67. P.1739. 10.
11. Cohen M., Emmerson B. Rheumatology of gout. 1994. V. 7. P. 1-16
12. Xosla U.M. et al. Hyperuricemia causes endothelial dysfunction. // Kidney lnternationa. 2005. Volume 67. P.1739.
13. Faschini F. Et al. Correlation between insulin-mediated glucose uptake and uric acid concentration in urine // SAMA. 1991. № 266. P. 3008-3011.
14. Fam A.G. gout nephropathy duo and insulin resistance syndrome // J. Revm. 2002. Volume 29. P. 1350-1355
15. Shostak N.A., Anichkov D.A. Metabolic syndrome: diagnostic criteria and the possibility of hypertensive therapy // Russian Medical Journal. 2002. № 27. pages 1258-1261
16. Shcherbak A.V., Balkarov I.M., Kozlovskaya L.V. Other fibrinological activity of urine as an indicator of renal injury with impaired uric acid metabolism. // Ter. Archive. 2001. T. 73 (6). S. 34-37.
